The MD Anderson researchers conclude that the ability to EZH2 to act genes that are needed to of cancer of cancer governs silence. If Akt is activated, it phosphorylates EZH2, so to rid it of a specific histone H3 known. If it is not bound to H3, can not EZH2 methylate H3 thus these silenced genes are re-expressed. If Akt is not activated, it is not phosphorylate EZH2, and this enzyme remains bound to and methylates H3, so that they silence silence gene expression. Our findings suggest that the Akt methylation activity of regulated through phosphorylation of EZH2, which may contribute to oncogenesis, Hung says..
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